Now, I realize that what I did was a “shock” routine; which broke me out of a rut. Now, as a regular schedule, I’d recommend training each bodypart with limited sets only every 4 to 5 days. But what I did worked. I had experimented with steroids at 18 for a few months; but never took them again. Never since before I turned 19. This “shock” routine worked BETTER than when I took that bottle of D-ball my firend at the gym had sold me. I did this routine at home. I made my own pully with rope from the hardware store and I made a “Scott” bench. I had 2, 4×4’s attached to a cross- board nailed to the studs up near the ceiling. The 4×4’s were about 40 inches apart and came down to the floor at a high angle. I had large holes drill about half-way into the 4×4’s so that I could place large bolts into them at different heights. I could also lay another 4×4 across, which I had rounded off and padded, and get between the wall and the rails to do my “Scott” curls. I put another board across the rails (high up) to attach a pully to. That was years ago; so you’ve come way too late to tell me that a natural guy can’t build big, cut arms. I did it; and most of that extra size lasted years afterwards, when I went back to normal training (though not as cut and impressive as when I followed this routine [my arm muscles didn’t “pop out” as much]). And I never took a steroid after my 18th year. I don’t mean to sound flippant; but “I KNOW” it can be done. You can get your arms past 16 inches naturally. I’m glad I didn’t read this article in a magazine years ago. I might not have even tried.
Flutamide acts as a selective, competitive , silent antagonist of the androgen receptor (AR).  Its active metabolite , 2-hydroxyflutamide , has between 10- to 25-fold higher affinity for the AR than does flutamide, and hence is a more powerful antiandrogen in comparison.     However, at high concentrations, unlike flutamide, 2-hydroxyflutamide is able to weakly activate the AR.   Flutamide has far lower affinity for the AR than do steroidal antiandrogens like spironolactone and cyproterone acetate, and it is a relatively weak antiandrogen in terms of potency by weight, but the large dosages at which flutamide is used appear to compensate for this.  In accordance with its selectivity for the AR, flutamide possesses no progestogenic , (direct) estrogenic , glucocorticoid , or antigonadotropic activity.   Similarly to nilutamide, bicalutamide, and enzalutamide , flutamide crosses the blood-brain-barrier and exerts central antiandrogen actions.